Skip to main content
. 2016 Apr 7;2016:3918219. doi: 10.1155/2016/3918219

Table 2.

Antioxidant defense system in the liver of Sprague Dawley rats with hepatotoxicity induced by CCl4 treated with Tamarindus indica L. tablets.

Group MDA SOD CAT GSH
(nmol/g tissue) (UI/mg tissue) (UI/mg tissue) (mg/g tissue)
I 0.47 ± 0.04c 0.35 ± 0.02a 0.40 ± 0.02a.b 29.74 ± 1.98b
II 1.49 ± 0.18a 0.15 ± 0.01d 0.13 ± 0.02d 15.84 ± 1.30c
III 0.66 ± 0.03b 0.31 ± 0.02b.c 0.36 ± 0.02c 32.58 ± 2.68a.b
IV 0.60 ± 0.04b.c 0.28 ± 0.03c 0.43 ± 0.02a 33.01 ± 2.26a
V 0.64 ± 0.03b 0.33 ± 0.02a.b 0.37 ± 0.03b.c 29.93 ± 0.90b

MDA: malondialdehyde; SOD: superoxide dismutase; CAT: catalase; GSH: reduced glutathione.

I: control group; II: CCl4 induced hepatotoxicity group; III: Silymarin treated group; IV and V: Tamarindus indica L. tablets treated groups at doses 100 mg/kg and 200 mg/kg, respectively.

Data are expressed as mean ± SD (n = 7).

Different letters on columns indicate significant statistical differences (p < 0.05).