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. 2016 Jan 27;6(1):1–8. doi: 10.1556/1886.2015.00048

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© The Author(s)

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 4. — DEC-HA treatment leads to the de novo generation of Foxp3⁺ Treg in VILLIN-HA transgenic mice. HA-specific CD4⁺Foxp3^– T cells were adoptively transferred into DEC-HA-, GL117-HA-, or PBS-treated VILLIN-HA transgenic mice. On day 6 posttransfer, spleens, MLN, and cells of the colonic lamina propria (LP) were analyzed by flow cytometry for the expression of the transgenic TCR and intracellular Foxp3. Data from three independent experiments are shown as means ± SEM. Statistical analysis was performed using one-way ANOVA (^**p < 0.01; ^***p < 0.001)