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The molecular pathways involved in the KKS induced vascular permeability. Bradykinin and DABK bind to B2R and B1R on endothelial cells. The activation of B1R and B2R increases the release of NO and PGI2 and VE-cadherin phosphorylation. PLA2: phospholipase A2; PGI2: prostacyclin; L-Arg: L-arginine; VEC: vascular endothelial cadherin.
