Skip to main content
. Author manuscript; available in PMC: 2016 Apr 26.
Published in final edited form as: Vascul Pharmacol. 2011 Mar 10;55(1-3):2–9. doi: 10.1016/j.vph.2011.03.001

Fig. 3.

Fig. 3

Role of potassium channels in alogliptin induced vasorelaxation. Mean cumulative relaxation of aortic rings in response to alogliptin after pre-incubation with A. TEA (6 μM), B. LNMMA, TEA and combined LNMMA and TEA, C. Charybdotoxin (100 nM), Apamin (100 nM), and combination of LNMMA, Charybdotoxin, and Apamin D. 4AP (1 mM). Results are presented as mean±SE; n=4–5/intervention; p<0.05 versus control for peak relaxation.