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. 2016 Apr 29;14:108. doi: 10.1186/s12967-016-0856-2

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© Yang et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Expression levels of deregulated miR-7, miR-93, miR-141, miR-195, miR-494, and let-7b in human CRC tumors. The relative expression level of miR is represented by log₁₀ (2^−ΔCt); ΔCt = (Ct_miR−Ct_U6b), with U6b as the internal control for normalization. a The samples from early relapsed patients showed significantly increased miR-7 expression levels (P = 0.014). b The samples from early relapsed patients showed significantly decreased miR-93 expression levels (P < 0.0001). c The samples from early relapsed patients showed significantly increased miR-141 expression levels (P = 0.001). d The samples from early relapsed patients showed significantly decreased miR-195 expression levels (P = 0.040). e The samples from early relapse patients showed significantly decreased miR-494 expression levels (P = 0.016). f The samples from early relapsed patients showed significantly decreased let-7b expression levels (P = 0.002)