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. Author manuscript; available in PMC: 2016 May 2.
Published in final edited form as: J Neurotrauma. 2012 Feb 29;29(8):1626–1637. doi: 10.1089/neu.2011.2190

FIG. 5.

FIG. 5

The anti-α4 treatment decreases the production of free radicals and lipid peroxidation in the kidney and liver at 24 h after spinal cord injury (SCI). (A and C) The concentration of 2′-7′-dichlorofluorescein (DCF) was assayed as a free radical marker in the kidney (A) and liver (C) homogenates from the uninjured (n = 6), T4C (n = 4), and T4T (n = 5) rats. (B and D) Lipid peroxidation was assessed by the thiobarbituric acid reactive substance (TBARS) assay for aldehydes, including malondialdehyde, in lung homogenates from the same groups of rats (group numbers the same as those in A and C; *significantly different from uninjured; #significantly different from T4 SCI controls; + tended to differ from T4 controls; p = 0.063; U, uninjured rats; T4C, T4 control SCI rats; T4T, T4 SCI rats treated with the anti-α4 monoclonal antibody [mAb]).