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. 2016 Mar 11;15(3):522–530. doi: 10.1111/acel.12461

Figure 1.

Figure 1

Endothelium‐dependent, nitric oxide (NO)‐dependent, and endothelium‐independent dilation. (A) Dose–responses to the endothelium‐dependent dilator acetylcholine (ACh) in young and old control (YC and OC) and young and old NMN‐supplemented (YNMN and ONMN) mice (n = 13–22 per group). (B) NO‐dependent dilation (Max DilationACh − Max DilationACh + l‐NAME) (n = 5–12 per group). (C) Dose–responses to the endothelium‐independent dilator sodium nitroprusside (SNP) (n = 7–22 per group). Values are mean ± SEM. *P < 0.05 vs. all.