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. 2016 May 4;14:114. doi: 10.1186/s12967-016-0869-x

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© Liu et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — Prdx1 inhibition blocked hypoxia/reoxygenation-induced IL-6 secretion. After hypoxia treatment for 6 h, control rabbit IgG (10 μg/mL) and Prdx1 antibody (2.5 and 10 μg/mL) were added separately to human monocytes under normoxia condition for another 24 h before the supernatants were collected and analyzed for IL-6 levels. Data represented means and SEM for triplicate samples. ***P < 0.001