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. 2016 May 4;14:116. doi: 10.1186/s12967-016-0847-3

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© Li et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — VEGF-B improved heart function in a myocardium I/R model. a TTC-stained myocardium 24 h after reperfusion. The white arrows indicate the infarct area of heart. b Infarction size in each group. The infarct size was expressed as a percentage of the left ventricular size for each heart. *P < 0.01 vs. sham group; ^# P < 0.01 vs. I/R group, n = 6. c Typical image of heart function. 24 h after reperfusion, LV function was measured as described in the “Methods” section. LVSP-left ventricle systolic pressure (d); LVEDP-left ventricle end-diastolic pressure (e); ±dp/dt _max-rate of the rise or fall of left ventricular pressure (g and h). *P < 0.01 vs. sham group; ^# P < 0.05 vs. I/R group; ^& P < 0.05 vs. VEGF-B (1.0) group, n = 6