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. Author manuscript; available in PMC: 2016 Oct 1.
Published in final edited form as: Biochim Biophys Acta. 2015 May 31;1853(10 Pt A):2404–2410. doi: 10.1016/j.bbamcr.2015.05.032

Figure 2. Effect of exogenous EGF and gp91 ds-tat on Mesenteric resistance arteries reactivity (n=6).

Figure 2

Percentage of contraction to thromboxane analogue (U46619) and phenylephrine (PE) and endothelium dependent relaxation to acetylcholine (ACh) in MRA from control (A, B, C) and type 2 diabetic mice (D, E, F) incubated with or without exogenous EGF and gp91 ds-tat. *P< 0.05 for Control vs. Control EGF. $P< 0.05 for Control vs. Control EGF+ gp91 ds-tat. ˆP< 0.05 for db−/db− vs. db−/db− + EGF and db−/db− + EGF + gp91 ds-tat.