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. 2016 May 1;24(3):328–337. doi: 10.4062/biomolther.2015.152

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Copyright ©2016, The Korean Society of Applied Pharmacology

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licens-es/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 8. — Effects of WO administration on the expression of brain-derived neurotrophic factor (BDNF) and cAMP-response element-binding protein (CREB) mRNAs in rats following TMT-induced hippocampal impairment. PCR bands on agarose gel and their relative intensities are indicated in (A) and (B), respectively. The expression levels of BDNF and CREB mRNAs were normalized to that of glyceraldehyde 3-phosphate dehydrogenase (GAPDH) mRNA as an internal control. ^*p<0.05 vs. the SAL group; ^#p<0.05 vs. the TMT group.