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. 2016 May 10;7:548. doi: 10.3389/fpls.2016.00548

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Copyright © 2016 Hossain and Dietz.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Circuitry of redox and ROS-related events in salinity stress response. Salinity stress causes metabolic imbalances and activates signaling pathways. The metabolic imbalances increase the ROS generation e.g., by enhanced photorespiration or cause overreduction of PET and RET as described in the review. The latter is modulated by AOX, PTOX and malate valve. Signaling activates RBOH. Accumulating ROS either cause excessive oxidation reactions leading to damage or via redox- and ROS-dependent signaling and regulation allow for proper acclimation.