Table 1.
Selected Clinical Trials of Gene Therapy Products for Critical Limb Ischemia
Reference (Trial) | Product | Dosage | Delivery Method | n | Rutherford Class | Follow-Up Duration | Outcomes: Bioactivity Parameters Improved |
---|---|---|---|---|---|---|---|
Fibroblast growth factor | |||||||
Belch et al55 (TAMARIS) | FGF-1; plasmid | 8×0.5 mg injections for 4 sessions | IM | 525 | Class 5–6 | 12 mo | No changes in major amputation or death rates |
Comerota et al152 | FGF-1; plasmid | Dose escalation; 1×500, 1×1000, 1×2000, 1×4000, 1×8000, 1×16 000 mcg; or 2×500, 2×1000, 2×4000, 2×8000 mcg | IM | 51 | Class 4–6 | 6 mo | Decrease in pain, aggregate ulcer size; Increased TcPO2, ABI |
Nikol et al153 (TALISMAN-201) | FGF-1; plasmid | 8×0.5 mg injections for 4 sessions | IM | 125 | No option CLI | 12 mo | Decreased amputation rate; no changes in ulcer healing |
Lederman et al56 (TRAFFIC) | FGF-2; recombinant | 30 mcg/kg single dose or 30 mcg/kg double dose (day 1+day 30) | IA | 190 | Class 2–3 | 3 mo | Increase peak walking time at 90 days; no change in quality of life or ABI |
Vascular endothelial growth factor | |||||||
Rajagopalan et al58 (RAVE) | VEG121; adenovirus | Low dose 4×109 pu; high dose 4×1010 pu; 20 injections | IM | 105 | Class 1–3 | 6 mo | No changes in peak walking time or ABI; increased peripheral edema |
Baumgartner et al154 | VEGF165; plasmid | 4000 mcg | IM | 9 | Class 4–6 | 10 wk | Increased vascularity on digital subtraction angiography (DSA); healing of ischemic ulcers; proliferation of endothelial cells on pathology |
Makinen et al155 | VEG165; plasmid and adenovirus | 2×1010 pfu VEGF-Ad; or 2 mg/2mL VEGF plasmid | IA | 56 | Class 1–6 | 3 mo | Increased vascularity on DSA |
Kusumanto et al156 | VEGF165; plasmid | 2 mg each for 2 session | IM | 54 | Class 4–6 | 100 days | No changes in amputation rates or rest pain; improvements in skin ulceration, ABIs, TBIs |
Hypoxia-inducible factor 1-alpha | |||||||
Rajagopalan et al60 | HIF-1alpha; adenovirus | Dose escalation; 1×108 to 2×1011 viral particles | IM | 34 | No option CLI | 12 mo | No serious adverse events attributable to study treatment; no amputations in 2 highest dose groups; complete rest pain resolution in 14 of 32 patients and complete ulcer healing in 5 of 18 patients |
Creager et al59 | HIF-1alpha; adenovirus | 2×109; 2×1010; 2×1011 viral particles; or placebo | IM | 289 | Class 1–3 | 12 mo | No significant differences in peak walking time, claudication onset time, ABI, or quality of life |
Developmental endothelial locus-1 | |||||||
Grossman et al157 | Del-1; plasmid | 42 mg plasmid or placebo over 21 injections in each leg (84 mg per subject) | IM | 105 | PAD and stable exercise-limiting IC | 12 mo | Improvement in peak walking time, ABI, claudication onset time, and quality of life but no difference between control group |
Hepatocyte growth factor | |||||||
Morishita et al63 | HGF; plasmid | Either 2 or 4 mg (4 or 8 injections) | IM | 22 | Fontaine IIb, III, or IV, | 2 yr | ABI, pain relief, and ulcer healing were improved in the majority of patients |
Powell et al158 (HGF-STAT) | HGF; plasmid | 0.4 mg×8 for 3 sessions (low); 4 mg×8 for 2 sessions (mid); 4 mg×8 for 3 sessions (high) | IM | 104 | Class 4–6 | 12 mo | Increased TcPO2 at 6 mo in high-dose group; no changes in amputation, death, ulcer size, wound healing, ABI, TBI |
Powell et al159 (HGF-0205) | HGF; plasmid | 0.5 mg×8 for 3 sessions | IM | 27 | Class 5–6 | 6 mo | No change in wound healing or amputation; increase in TBI at 6 mo; increase rest pain improvement |
Shigematsu et al160 | HGF; plasmid | 0.5 mg×8 for 2 sessions | IM | 40 | Class 4–5 | 3 mo | Increased reduction in ulcer size; increased quality of life; no change in rest pain or ABI |
Henry et al161 | HGF (VM202); plasmid | Dose escalation 2–16 mg | IM | 12 | No option CLI | 12 mo | Increase in median ABI/TBI; decrease in pain (visual analog scale) |
Gu et al162 | HGF (VM202); plasmid | 4 mg; 8 mg; 12 mg; 16 mg | IM | 21 | Class 4–6 | 3 mo | Increase in mean ABI and TcPO2; decrease in pain; improved wound healing |
ABI indicates ankle brachial index; Del-1, developmental endothelial locus-1; CLI, critical limb ischemia; FGF, fibroblast growth factor; HGF, hepatocyte growth factor; HIF-1α, hypoxia-inducible factor-1 alpha; IA, intra-arterial; IC, intermittent claudication; IM, intramuscular; PAD, peripheral artery disease; TBI, toe brachial index; TcPO2, transcutaneous oxygen pressure; and VEGF, vascular endothelial growth factor.