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. 2016 May 3;39(5):426–438. doi: 10.14348/molcells.2016.0094

Fig. 7.

Fig. 7.

A model of AVR-Pii and Os-NADP-ME2-3 in M. oryzae–rice interactions. Specific binding affinity of AVR-Pii with Os-NADP-ME2-3 suppresses Os-NADP-ME2-3 and inhibits NADPH production activity near the BIC. In the absence of AVR-Pii, active Os-NADP-ME2-3 catalyzes malic acid into pyruvic acid, generating NADPH, and subsequent production of apoplastic ROS with increased resistance to M. oryzae. In AVR-triggered ROS inhibition, secretion of AVR-Pii helps to establish compatibility in susceptible rice cultivar. EIHM, extra-invasive hyphal membrane; IH, invasive hyphae.