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. 2016 May 23;133(21):2050–2065. doi: 10.1161/CIRCULATIONAHA.115.021019

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© 2016 The Authors.

Circulation is published on behalf of the American Heart Association, Inc., by Wolters Kluwer. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 7. — Uptake of [¹⁸F]fluoride and [¹⁸F]FDG within plaque and normal artery and changes in noncoding RNA expression within carotid plaques. Axial images demonstrating unilateral (A, B) or bilateral [¹⁸F]fluoride carotid uptake (D, E). C is a multiplanar reformat of B. Axial images demonstrating [¹⁸F]FDG carotid uptake (F, G). H shows the Siemens Biograph Clinical PET/CT system used for imaging. White arrows indicate carotid radioligand uptake. H through K, L, Uptake of tracer: MicroRNA profile of atherosclerotic plaque and paired healthy carotid controls (n=6) assessed by qRT-PCR (paired Student t test). Expression of SMILR (M), HAS2 (N), and HAS2-AS1(O) within atherosclerotic plaque (n=5). Analyzed via qRT-PCR analysis, ***P<0.001, **P<0.01, and *P<0.05 assessed by paired Student t test. CT, computed tomography; [¹⁸F]FDG, ¹⁸F-fluorodeoxyglucose; PET, positron emission tomography; and qRT-PCR, quantitative real-time polymerase chain reaction.