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. 2016 Jan 19;7(6):7343–7353. doi: 10.18632/oncotarget.6944

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Copyright: © 2016 Singh et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Untreated (), CXCL16 treated with () or without CXCR6 blockade () were incubated with DilC₁₂ labeled human bone marrow endothelial cells (HBME) cells for 30 min. Attached cells were counted by measuring fluorescence of DilC₁₂ (3) at 549/565nm (Ex/Em) after washing off the unattached HBME cells. Results are expressed as mean ± SE. Asterisk (^*) indicates significant difference (p < 0.05) in PCa cell adhesion between treated and control cells.

Inline graphic — Untreated (), CXCL16 treated with () or without CXCR6 blockade () were incubated with DilC₁₂ labeled human bone marrow endothelial cells (HBME) cells for 30 min. Attached cells were counted by measuring fluorescence of DilC₁₂ (3) at 549/565nm (Ex/Em) after washing off the unattached HBME cells. Results are expressed as mean ± SE. Asterisk (^*) indicates significant difference (p < 0.05) in PCa cell adhesion between treated and control cells.