Skip to main content
. Author manuscript; available in PMC: 2017 May 1.
Published in final edited form as: Cancer Epidemiol Biomarkers Prev. 2016 Mar 14;25(5):780–790. doi: 10.1158/1055-9965.EPI-15-1039

Table 2.

Association with p < 10−4 for GE and BMI-GE analyses. Results highlighted in green or red in Figures 1 and 3. More detailed summaries of these top hits are shown in Supplemental Tables 4 and 5.

Analysis Histology Environment Gene SNP MAF N cases N controls Interaction P-value
GE All EPP MHT IGF1R rs41497346 0.28 2870 5051 −0.577 4.92 × 10−6
GE All Endometriosis ESR1 rs12661437 0.34 5335 9306 0.534 1.47 × 10−5
GE ENDO Estrogen MHT HSD17B2 rs2955162 0.23 405 5054 −1.12 3.44 × 10−5
GE HGS Endometriosis CYP11A1 rs9944175 0.24 2578 9306 −0.872 4.13 × 10−5
GE ENDO Estrogen MHT AKR1C3 rs61856140 0.10 405 5054 −1.47 5.30 × 10−5
GE ENDO Endometriosis CYP11B2 rs28526467 0.43 815 9306 −1.09 6.83 × 10−5
GE ENDO OC Use PRL rs72836169 0.10 945 10323 −0.827 7.09 × 10−5
BMI-GE ENDO Parity INSR rs8102954 0.37 778 8284 −2.56 8.35 × 10−6
BMI-GE All Parity IGFBP2 rs869564 0.11 4934 8284 −2.34 1.43 × 10−5
BMI-GE HGS OC Use CYP11B1 rs113759408 0.17 2296 8250 1.49 2.18 × 10−5