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. 2016 Jan 8;7(4):e1118195. doi: 10.1080/19382014.2015.1118195

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© 2015 The Author(s). Published with license by Taylor & Francis Group, LLC

This is an Open Access article distributed under the terms of the Creative Commons Attribution-Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The moral rights of the named author(s) have been asserted.

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Figure 4. — PAR3-AP effects in human islets and thrombin generation in T2D patients. (A) Insulin secretion in human islets (n=4 −8). (B) Increase in cell capacitance (n = 11–12). (C) Accumulated insulin levels following 2, 12 or 24-h incubations of islets from non-diabetic Wistar rats in medium supplemented with plasma from Wistar rats (Ctrl) or ZDF rats with or without 25 μg/ml H103 (n = 5–10). (D–E) The lag time of thrombin activation plotted against insulinogenic index and fasting glucose. (F-G) The peak concentration of thrombin and endogenous thrombin potential (AUC), respectively, plotted against fasting insulin corrected for glucose (n = 17).