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. 2016 Apr 12;13(6):4606–4612. doi: 10.3892/mmr.2016.5108

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Copyright: © Fu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Inhibition of RUNX3 regulates the frequencies of Th17 and Th22 in CD4⁺ T cells from patients with psoriasis. The frequencies of Th17 and Th22 cells in CD4⁺ T cells from patients with psoriasis transfected with RUNX3 siRNA or an siRNA control was detected using flow cytometry. (A and B) Flow cytometric analysis of Th17 in CD4⁺ T cells from patients with psoriasis. The percentages of cells in the Q2 region represent the percentage of Th17 cells. (C and D) Flow cytometric analysis of Th22 in CD4⁺ T cells from patients with psoriasis. The percentages of cells in the Q2 region represent the percentage of Th22 cells. (E) Percentages of Th17 and Th22 cells in CD4⁺ T cells from patients with psoriasis transfected with RUNX3 siRNA or an siRNA control. Data are presented as the mean ± standard deviation of three experiments. ^*P<0.05, vs. control group. RUNX3, runt-related transcription factor 3; Th, T helper; IL, interleukin; siRNA, small interfering RNA.