Table 2.
Gene ontology (GO) enrichment of consensus ROH analysisa
GO: ID | Go: term |
---|---|
GO:0006915 | Apoptotic process |
GO:0002209 | Behavioral defense response |
GO:0001662 | Behavioral fear response |
GO:0070588 | Calcium ion transmembrane transport |
GO:0071345 | Cellular response to cytokine stimulus |
GO:0071495 | Cellular response to endogenous stimulus |
GO:0071396 | Cellular response to lipid |
GO:0033554 | Cellular response to stress |
GO:0019221 | Cytokine-mediated signaling pathway |
GO:0006281 | DNA repair |
GO:0007631 | Feeding behavior |
GO:0007599 | Hemostasis |
GO:0031663 | Lipopolysaccharide-mediated signaling pathway |
GO:0032873 | Negative regulation of stress-activated MAPK cascade |
GO:0070303 | Negative regulation of stress-activated protein kinase signaling cascade |
GO:0016310 | Phosphorylation |
GO:0042981 | Regulation of apoptotic process |
GO:0080135 | Regulation of cellular response to stress |
GO:0001959 | Regulation of cytokine-mediated signaling pathway |
GO:0043408 | Regulation of MAPK cascade |
GO:2000377 | Regulation of reactive oxygen species metabolic process |
GO:0080134 | Regulation of response to stress |
GO:0032319 | Regulation of rho gtpase activity |
GO:1901700 | Response to oxygen-containing compound |
GO:0009314 | Response to radiation |
GO:0006950 | Response to stress |
GO:0023014 | Signal transduction by phosphorylation |
GO:0033209 | Tumor necrosis factor-mediated signaling pathway |
GO:0042060 | Wound healing |
GO:0003684 | Damaged DNA binding |
GO:0005246 | Calcium channel regulator activity |
GO:0005488 | Binding |
GO:0008289 | Lipid binding |
GO:0030234 | Enzyme regulator activity |
GO:0005509 | Calcium ion binding |
GO:1901363 | Heterocyclic compound binding |
GO:0005160 | Transforming growth factor beta receptor binding |
GO:0005543 | Phospholipid binding |
GO:0004674 | Protein serine/threonine kinase activity |
GO:0035258 | Steroid hormone receptor binding |
GO:0008083 | Growth factor activity |
Subset of GO terms that could putatively affect responses to environmental stressors. aAll terms were statistically significant at FDR < 0.15. This analysis lends evidence to support that all three populations experienced selective pressures for variants effecting stress response, immune response, and behavior