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. 2016 May 26;20:157. doi: 10.1186/s13054-016-1344-z

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Distribution of classification scores of the AKI marker pattern (upper panel) and NGAL levels in ng/mL (lower panel) in AKI and non-AKI patient subgroups showing either progression or lack of nonocclusive mesenteric ischemia (NOMI) or death. A post hoc test was performed for average rank differences between the different subgroups (each with p < 0.05) after a significant result in the global Kruskal-Wallis test (p < 0.0001). AKI acute kidney injury, NGAL neutrophil gelatinase-associated lipocalin