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. 2016 May 28;9:61. doi: 10.1186/s13041-016-0237-z

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© Lillenes et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — PARP1, APE1 and Polβ protein levels are modified in AD. Quantitative protein detection was performed by mass spectrometry (nLC and Thermo Q Exactive). Levels of base excision repair proteins PARP1, APE1 and Polβ were compared in Alzheimer’s patients (AD) and healthy controls (HC). Reference to house-keeping proteins is shown in Additional file 1: Figure S1. Black bars: PARP1, grey bars: APE1, light grey bars: Polβ. DNA glycosylase OGG1 was below the detection limit. Abbreviations: FC = Frontal cortex, CB = Cerebellum