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. 2016 Jan 9;7(7):7856–7865. doi: 10.18632/oncotarget.6856

Figure 2. GPR171 promotes proliferation of lung cancer cells.

Figure 2

A. Cell viability assays of Calu-6 lung cancer cells expressing control or two different GPR171 shRNAs (n = 3; **P < 0.01, ***P < 0.001 vs. control shRNA, Student's t test). B. Cell viability assays of A549 lung cancer cells expressing control or two different GPR171 siRNAs. Results are presented as means ± SEM (error bars) (n = 3; **P < 0.01, ***P < 0.001 vs. control siRNA, Student's t test). C. Cell viability assays of WI-38, and IMR-90 cells expressing control vector or a GPR171 expression plasmid. Results are presented as means ± SEM (error bars) (n = 3; *P < 0.05, **P < 0.01 vs. vector control, Student's t test). Error bars on the values were smaller than the symbols. Knockdown or ectopic expression of GPR171 was confirmed by immunoblotting (insets). Tubulin was used as a loading control.