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. 2016 Feb 1;48(3):257–265. doi: 10.1093/abbs/gmv137

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© The Author 2016. Published by ABBS Editorial Office in association with Oxford University Press on behalf of the Institute of Biochemistry and Cell Biology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences.

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Figure 5. — OSM receptor Oβ knockout (Oβ-KO) attenuated DCM injury (A) Representative images as evaluated by echocardiography. (B) TEM showed relieved mitochondria impairment in the DCM-Oβ^−/− group and OSM could no longer lead to mitochondria impairment in DCM-Oβ^−/− group mice (scale bar = 2 μm). (C) Sirius red staining showed decreased fibrosis area in the DCM-Oβ^−/− group compared with the DCM group, and no increased fibrosis area was detected in the DCM-Oβ^−/− +OSM group (magnification, ×400). (D–G) Statistical analysis of EF, FS, ±LV dp/dt max, LVEDV and LVESV. (H) Statistical analysis of fibrosis area. *P < 0.05 vs. DCM group.