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. Author manuscript; available in PMC: 2017 Jun 15.
Published in final edited form as: J Neuroimmunol. 2016 Apr 16;295-296:68–74. doi: 10.1016/j.jneuroim.2016.04.005

Figure 3.

Figure 3

Performance on the rotarod (as a percentage of the pre-stroke baseline) is depicted on the Y-axis and data presented as the median (IQR). Infection worsened outcomes in animals treated with ceftiofur, but only when the antibiotic was given concomitant with infection (a). Infection did not worsen outcomes in enrofloxacin treated animals, and delay of enrofloxacin was associated with better outcomes (b). Among ceftiofur treated animals, a delay in antibiotics was associated with a better outcome in Staphylococcus aureus (SA) infected animals (c). For enrofloxacin treated animals, a delay in antibiotics was associated with a better outcome in Pseudomonas aeruginosa (PA) infected animals (d), PA infection was generally associated with worse outcomes than SA infections (d). Statistics are by Kruskal-Wallis H test (a and b) and differences among the three groups noted by *(P<0.05) or **(P<0.01). Pathogen related differences for either immediate or delayed antibiotics are assessed by Mann-Whitney U-test (c and d) and differences indicated by †(P<0.05) or ‡ (P<0.01). For a given pathogen, differences based on antibiotic timing are assessed by Mann-Whitney U-test and indicated by *(P<0.05) or **(P<0.01).