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. 2016 Jan 30;11:8. doi: 10.1186/s13013-016-0063-1

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© Burwell et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Cascade Concept for AIS pathogenesis based on the findings of Clark et al. [4] that place adipose tissue and energy control in relation to the predisposition to AIS. It is speculated that leptin is linked to human central nervous system development, asynchronous neuro-osseous growth mechanism, and the dorsal shear mechanism. NCS = neurocentralsynchondrosis, GP = growth plate