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. 2016 Jun;26(6):852–862. doi: 10.1101/gr.203992.116

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© 2016 Blango and Bass; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genome.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 4. — A subset of mEAGs also have EERs in the human ortholog. (A) Intersection of mEAGs with hEAGs (P < 0.0001 by χ² test). (B) Primary intersecting feature of conserved EAGs defined as 3′ UTR, Intron, or Other. Gene structure, EER location, base-fraction editing (red, BFE 0%–100%), and relative expression (blue, FPKM, 0–10 reads) are shown for mouse (C) and human (D) Sppl2a/SPPL2A orthologs. Data are for 6 h post-LPS treatment of mouse BMDMs and human PBMs. Mfold-predicted EER structures are compared to the right (E,F).