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. 2016 May 1;9(5):501–512. doi: 10.1242/dmm.023416

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© 2016. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 3. — UVRAG loss impairs gut function and survival. (A) Stem- and progenitor-cell-specific knockdown of Uvrag in the intestine leads to increased retention of ingested Bromophenol-Blue-containing food (a constipation-like phenotype). Error bars represent s.d. (B) Loss of UVRAG in ISCs and EBs decreases lifespan. (C) Treatment with the toxin DSS kills UVRAG RNAi flies much faster than control flies. (D) Animals undergoing Uvrag knockdown in esg-positive cells are hypersensitive to infection by Pseudomonas aeruginosa.