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. 2016 May 25;36(6):1174–1185. doi: 10.1161/ATVBAHA.115.306983

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© 2016 The Authors.

Arteriosclerosis, Thrombosis, and Vascular Biology is published on behalf of the American Heart Association, Inc., by Wolters Kluwer. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 2. — Aorta transcript maps reveal the specificity and territoriality of B-cell–related immune responses in artery tertiary lymphoid organs (ATLOs). A, Heatmaps of differentially expressed genes in the adventitia cluster (wild-type [WT], n=3; ApoE^−/−, n=4). B, Plaque/ATLO cluster (for plaque, n=3; for ATLOs, n=4). C, Lymph node (LN) cluster (for WT and ApoE^−/− LNs, n=3; for ATLO, n=4); gene ontology terms immune system process, B-cell activation, B-cell–mediated immunity, immunoglobulin, positive regulation of B-cell–mediated immunity, positive regulation of B-cell activation, B-cell differentiation, and B-cell proliferation. D, Selected genes in the LN cluster. Results represent mean±SEM. Analyses were performed using ANOVA with Benjamini–Hochberg correction. Absolute numbers of signal intensities and statistics are reported in Table I in the online-only Data Supplement. RLN indicates renal LNs.