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. 2016 Jun 7;6:26992. doi: 10.1038/srep26992

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Blast induced bradycardia as a function of peak overpressure (A) and impulse (B). In both cases, the dose-response function (1) was used to model the pathological response and resulting parameters are listed in respective insets in both figures. All blast exposed groups have statistically significant heart rate decrease versus control (p < 0.05).