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. Author manuscript; available in PMC: 2017 Jul 1.
Published in final edited form as: Hypertension. 2016 May 9;68(1):227–235. doi: 10.1161/HYPERTENSIONAHA.116.07232

Table 1.

Neurohormonal and Metabolic Responses to Baroreflex Activation and Carotid Body Denervation

CONDITION PRA (ng ANG I/mL/hr PALDO (ng/dL) PCORT (μg/dL) PNE (pg/mL) PINS (μU/mL) PGLU (mg/dL) BW (kg)
CONTROL 1.0±0.1 5.5±0.8 2.0±0.4 121±36 8.2±0.8 108±5 24.7±0.3
HIGH-FAT 2.6±0.6 7.4±1.5 2.5±0.5 132±23 20.1±2.5* 93±5 33.8±1.2
REDUCED-FAT 1.7±0.5 5.6±0.5 2.9±0.8 139±23 17.2±2.3* 106±3 33.7±1.4
BA 1.0±0.3† 4.3±0.6 2.5±0.4 83±8*† 16.0±1.2* 107±4 33.7±1.1
RECOVERY 1.2±0.2 4.6±0.4 2.0±0.6 112±15 17.4±2.7* 110±3 33.6±1.2
CBD 1.2±0.2 5.0±0.6 2.3±0.3 110±4 19.7±2.8* 106±2 33.7±1.1

Values are mean±SE: n=4. PRA indicates plasma renin activity; PALDO plasma aldosterone concentration; PCORT, plasma cortisol concentration; PNE, plasma norepinephrine concentration; PINS, plasma insulin concentration; PGLU, plasma glucose concentration. BA, baroreflex activation; CBD, carotid body denervation.

*

P<0.05 vs control,

†

P<0.05 vs reduced fat.