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. 2016 Mar 15;42(4):992–1002. doi: 10.1093/schbul/sbw022

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© The Author 2016. Published by Oxford University Press on behalf of the Maryland Psychiatric Research Center. All rights reserved. For permissions, please email: journals.permissions@oup.com

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Fig. 3. — Expression levels of GABA-associated mRNAs in the cortex of mice with glutamic acid decarboxylase 67 (GAD67) knockout in parvalbumin (PV) neurons. Mean ± SEM expression levels of GABA transporter-1 (GAT-1; A), PV (B), KCNS3 (C), brain-derived neurotrophic factor (BDNF; D), tropomyosin receptor kinase B (TrkB; E), GABA_AR α1 (F), somatostatin (SST; G), and glutamic acid decarboxylase 65 (GAD65; H) mRNAs in the prefrontal cortex (PFC) and sensorimotor cortex (SMC) of PV^GAD67+/+, PV^GAD67+/−, and PV^GAD67−/− mice at 8 and 15 weeks of age. Bars not sharing the same letter are statistically different.