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. 2016 Jun 14;9:8. doi: 10.1186/s13069-016-0045-1

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Phenotypes of mice carrying the deletion of Pten (Pten null) in the liver. a Body weight of control and Pten null mice. b Fasting plasma glucose in control and Pten null mice. n = 7–10. *p < 0.05. c Liver-specific Pten deletion mice develop fatty liver disease at an early age (top; FL fatty liver). Adenomas are also observed in these mice (middle, nl normal liver, Ad adenomas). Morphologies that resemble Von Meyenburg syndrome are also observed in the livers of the liver Pten null mice (bottom)