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. Author manuscript; available in PMC: 2016 Sep 16.
Published in final edited form as: Nature. 2016 Mar 16;531(7595):528–532. doi: 10.1038/nature17184

Extended Data Fig 2. Exacerbation of fat accumulation and tubular injury in post-ischemic PGC1α−/− kidneys.

Extended Data Fig 2

a–d, Low (a,b) and high-power (c,d) photomicrographs 24 h after IRI in WT vs. PGC1α−/− (KO) mice. Scale bars 100 and 50 μm. e,f, Blinded scoring of tubular injury in cortex and outer stripe of outer medulla (OSOM) on 4-point injury scale as described in Methods (n=8 WT vs.12 KO mice), *p<0.05. g, Di-/tri-acylglycerols (DAGs, TAGs) in renal homogenates of KO mice at baseline and 24 h after injury (n=6/group). Each bar represents one lipid species. P-value by two-way ANOVA. Error bars SEM.