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. Author manuscript; available in PMC: 2017 Jun 7.
Published in final edited form as: Sci Signal. 2016 Jun 7;9(431):ra59. doi: 10.1126/scisignal.aad3373

Fig. 5. GATA6L abundance is not altered by prolonged rapamycin treatment or feedback phosphorylation of Akt.

Fig. 5

(A) HT-29 cells exhibit rapamycin-induced feedback phosphorylation of Akt, but do not stabilize GATA6. a.u., arbitrary units. (B) AC16 cells exhibit stabilization of GATA6S, but not GATA6L, without rapamycin-induced feedback phosphorylation of Akt. Vinculin,, tubulin, and glyceraldehyde-3-phosphate dehydrogenase (GAPDH) used as loading controls (51). Quantitative immunoblot data are means ± SEM of n = 4 biological replicates across two separate experiments.