Table 6.
Correlations of responsea to infection with PRRSV isolate KS06 using the full G-matrix
| Trait | VL | WG | TP | PV | Tmax | Vmax |
|---|---|---|---|---|---|---|
| VL | −0.23 (0.05) | −0.06 (0.05) | 0.76 (0.02) | 0.13 (0.05) | −0.16 (0.05) | |
| WG | −0.52 (0.17) | −0.05 (0.05) | −0.13 (0.05) | −0.06 (0.06) | 0.13 (0.05) | |
| TP | −0.08 (0.22) | −0.10 (0.24) | 0.02 (0.05) | 0.80 (0.02) | 0.02 (0.05) | |
| PV | 0.91 (0.05) | −0.30 (0.18) | −0.08 (0.25) | −0.19 (0.05) | 0.52 (0.04) | |
| Tmax | 0.19 (0.23) | −0.42 (0.23) | 0.69 (0.19) | −0.24 (0.28) | −0.52 (0.04) | |
| Vmax | −0.01 (0.20) | 0.42 (0.21) | −0.12 (0.28) | 0.51 (0.13) | −0.75 (0.18) |
WG weight gain (kg), VL viral load (area under the Wood’s curve of log10 serum viremia from 0 to 21 days post infection; viremia * days), TP time to peak viremia (days), PV peak viremia (log10 serum viremia), Tmax time to maximal rate of viremia decay (days), Vmax maximal rate of viremia decay (log10 serum viremia/day)
aPhenotypic correlations (above diagonal) and genetic correlations (below diagonal) were estimated using an animal model in ASReml and the full G-matrix