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. Author manuscript; available in PMC: 2017 Aug 1.
Published in final edited form as: Brain Behav Immun. 2016 Mar 2;56:209–220. doi: 10.1016/j.bbi.2016.02.025

Fig. 2. Wet-cage stress exposure concurrently with CpG-C treatment abolished its beneficial effects on MADB106 LTR, both in the context of adrenergic challenge (metaproterenol) and without it.

Fig. 2

In the absence of prolonged stress (HCC), CpG-C decreased LTR levels both in animals administered with metaproterenol (simultaneously with tumor challenge) or with placebo (PBS) (*), whereas in animals subjected to the Wet cage stress condition, no effects of CpG-C were evident in either condition. * indicates a significant difference from PBS at p < 0.05. Data is shown as mean ± SEM.