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. 2016 Jun 24;6:28253. doi: 10.1038/srep28253

Table 1. LW/MW gene cluster composition and LW and MW exon 3 haplotypes in BCM and XLCD families analysed in this study.

Index subjects Exon 3
Clinical diagnosis
LW
MW
Haplotype p. Haplotype p. nd
ZD379-19194 1 LIAVA 2 MIAVAc.465C 2 BCM
BCM101-19818 1 LIAVA 3 MIAVAc.465G 2 BCM
BCM51-12359 1 LIAVA 9 MVVVAc.465G 2 XLCD
BCM160-23130 1 LIAVA 9 MVVVAc.465G 1 BCM
ZD314-18057 1 LIAVA 10 MVVVAc.465C 1 XLCD
BCM126-20616 4 LVAVA 4 LVAVA 1 BCM
BCM133-20961 4 LVAVA 5 LVAIA 1 BCM
ZD547-4544 4 LVAVA 12 MVAVA 1 XLCD
BCM98-19713 8 LIAIA 7 MVAIA 1 XLCD
BCM142-21958 6 LVAISc 5, 7 LVAIA, MVAIA 2 BCM
BCM72-17075a 1 LIAVA 1, 10 LIAVA, MVVVAc.465C 3 BCM
BCM72-16874a 11 LIAVS 1, 10 LIAVA, MVVVAc.465C 3 Deutan/Macular Dystrophy
BCM73-16953b 1 LIAVA 0 BCM/XLCD
BCM93-19164 1 LIAVA 0 XLCD
BCM66-16407 4 LVAVA 0 BCM
BCM112-23518b 4 LVAVA 0 CRDe
BCM194-25474 4 LVAVA 0 BCM

aAffected subjects from the same family with distinct genotypes.

bSubjects harbouring single LW/MW hybrid genes.

cThis haplotype LVAIS includes an additional missense variant (c.556C > T/p.P186S; RefSeq: NM_020061.5) in exon 3 of the LW gene.

dNumber of MW gene copies deduced from qPCR.

eCone-Rod Dystrophy due to additionally impaired rod function (see Supplementary Table S1).