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. 2016 Jun 28;15:90. doi: 10.1186/s12933-016-0406-5

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Immunohistochemical analysis for omentin-1 in human adipose tissue. A representative slides of adipose tissue from patients in the CAD group (Fig. 3 A-a, EAT of non-stenosis subgroup; 3A-b, EAT of stenosis subgroup; 3A-c, SAT) and the NCAD group (Fig. 3 A-d, EAT; 3A-e, SAT) (magnified ×200). B results of quantitative immunohistochemical analysis for omentin-1 in EAT and SAT of the two groups (CAD group, n = 9; NCAD group, n = 9). C results of quantitative immunohistochemical analysis for omentin-1 in EAT of CAD group (total, n = 18; stenosis subgroup, n = 9; non- stenosis subgroup, n = 9) and NCAD group (n = 9). *P < 0.05, **P < 0.01. CAD coronary artery disease, EAT epicardial adipose tissue, SAT subcutaneous adipose tissue, IOD integrated optical density