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. 2016 Jun 29;6:28754. doi: 10.1038/srep28754

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(a) Interruption of the oxidative branch of the pentose phosphate pathway (oxPPP) in mature human erythrocytes by the anti-inflammatory compounds Bay 11–7082, parthenolide and dimethyl fumarate (DMF). The susceptibility of erythrocytes to eryptosis mediated by these compounds is partially based on the inhibition of G6PDH and GR activities. (b) Use of G6PDH inhibitors Bay 11–7082, parthenolide and/or dimethyl fumarate (DMF) as important metabolic-based therapy for inflammatory diseases.