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. 2016 Jun 15;30(12):1440–1453. doi: 10.1101/gad.277178.115

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© 2016 Zhou et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 6. — Ino80 occupies oncogenic SE genes and promotes their expression. (A) Average ChIP-seq read density of Med1, H3K27ac, and H3K4me1 at Ino80-bound and Ino80-unbound genes. (B) Venn diagram showing the overlap between Ino80-bound and SE genes. (C) Box plots showing gene expression changes for all genes (ALL) versus SE genes upon Ino80 silencing in A375 cells at the indicated time points. The P-values were calculated by Student's t-test. (D) GSEA showing that SE genes were preferentially enriched for those that are down-regulated after Ino80 silencing.