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. 2016 Jun 29;16:51. doi: 10.1186/s12935-016-0330-5

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Observation of DNA fragmentation and westerblot analysis of apoptotic proteins in ETME treated A549 and MCF-7 cells. a The fragmented DNA of nuclei were bind with DAPI and observed under a confocal microscope (630×). The white arrows indicate cells with fragmented DNA of nucleus after the treatment of ETME. b Effect of ETME on apoptotic proteins in A549 (200 µg/ml) cells and c MCF-7 (150 µg/ml) cells analysed by immunoblotting after various time intervals. Expression of β-actin was set as a protein loading control