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. 2016 Jun 6;5(6):57. doi: 10.3390/jcm5060057

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© 2016 by the authors; licensee MDPI, Basel, Switzerland.

This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC-BY) license (http://creativecommons.org/licenses/by/4.0/).

PMC Copyright notice

Integrin αvβ3-targeted imaging of myocardial angiogenesis. Example of ex vivo ^99mTc-sestamibi (reflecting perfusion, (left)) and ¹¹¹In-RP748 (reflecting αvβ3 integrin expression and activation, (center)) and fused color-coded (right) images of canine myocardium at three weeks post myocardial infarction induced by left anterior descending coronary artery occlusion. Yellow arrows point to the margins of the perfusion defect, and white arrows show the area of increased ¹¹¹In-RP748 uptake. On fused images perfusion tracer uptake is in green. RP748 accumulation (in red) is localized in the areas of perfusion defect. Published with permission of the American Society for Clinical Investigation, from “Noninvasive imaging of myocardial angiogenesis following experimental myocardial infarction”, Meoli, D.F., et al. J. Clin. Invest. 2004, 113(12), 1684–1691 [38]; permission conveyed through Copyright Clearance Center, Inc.