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. 2016 Jul 1;24(4):347–362. doi: 10.4062/biomolther.2016.067

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Copyright ©2016, The Korean Society of Applied Pharmacology

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 8. — The mechanism of adrenergic receptors. A represent α-receptors and trigger the IP₃ (Inositol triphosphate) which then increase the Ca²⁺ level in cytoplasm and causing muscles contraction. B represents β-adrenoreceptors. The represents Marine compounds. Xestospongin C inhibit the phospholipase enzyme which play a key role in activation of IP₃ (Inositol triphosphate) and block Ca²⁺ channels. S1319 B-2 receptor agonist resulting Bronchodilation and uterus relaxation.

Inline graphic — The mechanism of adrenergic receptors. A represent α-receptors and trigger the IP₃ (Inositol triphosphate) which then increase the Ca²⁺ level in cytoplasm and causing muscles contraction. B represents β-adrenoreceptors. The represents Marine compounds. Xestospongin C inhibit the phospholipase enzyme which play a key role in activation of IP₃ (Inositol triphosphate) and block Ca²⁺ channels. S1319 B-2 receptor agonist resulting Bronchodilation and uterus relaxation.