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. Author manuscript; available in PMC: 2017 Jul 1.
Published in final edited form as: Alcohol Clin Exp Res. 2016 Jun 13;40(7):1430–1442. doi: 10.1111/acer.13116

Fig. 5.

Fig. 5

Transduction of activated HSCs with AAV2 miR19b increased miR19b expression and inhibited increases in pri-miR17-92. (A) GFP expression in primary HSCs transduced with either AAV2-control (AAV2-con) or AAV2-miR19b and exposed to conditioned media from HepG2E47 treated with 0 (E47CM0) or 25mM (E47CM25) EtOH. (B) miR19b expression in E47CM0 and E47CM25 cells transduced with AAV2 miR19b compared to cells transduced with AAV2 control (*p < 0.05). (C) pri-miR17-92 expression in E47CM25 exposed cells transduced with AAV2 control or AAV2 miR19b (*p<0.05 vs E47CM0; #p < 0.05 vs AAV2 Control with EtOH).