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. 2016 Jul 7;6:29041. doi: 10.1038/srep29041

Figure 7. Schematic diagram illustrating hypothetical mechanisms by which an elevation in [Ca2+]i as a result of TRPC6 channel activation, and subsequent induction of the calcineurin/NFAT, FasL/Fas, and caspase signaling cascades promote neonatal GMC apoptosis.

Figure 7

CASP (caspase); Ac-DEVD-CHO (A-D-C); BA (blocking antibody); CaM (calmodulin).