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. 2016 Apr 26;203(3):1235–1247. doi: 10.1534/genetics.115.186064

Table 2. AVG cross-over defects are rescued by an aex-3-containing fosmid, expression of the cDNA in AVG and unc-5 overexpression (% animals with defects).

Genotype AVG CO n
aex-3 fosmid (WRM0629Da08)
aex-3(hd148); nid-1; hdEx602(+) 24** 102
aex-3(hd148); nid-1; hdEx602(−) 50 ns 104
aex-3 cDNA
aex-3(hd148); nid-1; hdEx610(+) 18** 128
aex-3(hd148); nid-1; hdEx610(−) 45 ns 95
aex-3(hd148); nid-1; hdEx611(+) 16** 130
aex-3(hd148); nid-1; hdEx611(−) 48 ns 100
aex-3(hd148);nid-1; hdEx612(+) 13** 116
aex-3(hd148); nid-1; hdEx612(−) 47 ns 94
unc-5 overexpression
aex-3(hd148); nid-1; evIs98c 25** 132

Marker used: hdIs51[odr-2::tdTomato]. n = number of animals. For rescue experiments, animals with (+) and without (−) extrachromosomal arrays were counted. For the aex-3 rescue the following independent transgenic lines were analyzed: hdEx602 [WRM0629dA08; myo-2::GFP]; hdEx610, 611, and 612 [plin-11::aex-3 cDNA::GFP; unc-122]; and evIs98c [unc-5p::UNC-5::GFP]. For statistical significance, we compared transgenic lines with aex-3(hd148); nid-1 double mutant. ** P < 0.01; ns, not significant; χ2 test.