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. 2015 Jul 7;1(4):212–224. doi: 10.1002/cjp2.23

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© 2015 John Wiley and Sons Ltd and The Pathological Society of Great Britain and Ireland

This is an open access article under the terms of the Creative Commons Attribution‐NonCommercial‐NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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E and S components of sarcomatoid ccRCC show similar DNA copy number aberrations that differ from those of non‐sarcomatoid ccRCC. (A) Similar genome‐wide DNA copy number signature of E and S components of sarcomatoid ccRCC. (B) Distinct genome‐wide DNA copy number signature of sarcomatoid ccRCC versus advanced‐stage non‐sarcomatoid ccRCC (E*) TCGA cases. (C) Distinct genome‐wide DNA copy number signature of sarcomatoid ccRCC versus Fuhrman grade 4 non‐sarcomatoid ccRCC (E*) TCGA cases. Copy number alterations are mapped according to their chromosomal location on the x‐axis, with the y‐axis showing the percentage of cases that harbour these changes.