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. 2016 Jun 14;14(2):1523–1530. doi: 10.3892/mmr.2016.5401

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Copyright: © Deng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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(A) Cyclin-dependent kinase inhibitor 3 silencing downregulated PCNA, Bcl-2, vimentin and RhoA expression levels, and upregulated Bax expression levels in (B) MCF-7 and (C) BT474 cells. Data are presented as the mean ± standard deviation. ^*P<0.05; ^**P<0.01; ^***P<0.001 compared with the control group. PCNA, proliferating cell nuclear antigen; Bcl-2, B-cell lymphoma 2; Bax, Bcl-2-associated X protein; RhoA, Ras homolog gene family, member A; siRNA, small interfering RNA; GAPDH, glyceraldehyde 3-phosphate dehydrogenase.