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. Author manuscript; available in PMC: 2017 Aug 1.
Published in final edited form as: Pancreas. 2016 Aug;45(7):997–1002. doi: 10.1097/MPA.0000000000000643

Table 3.

Demographics, patterns of alcohol use, cigarette smoking and clinical outcomes for sentinel episode of AP by alcoholic etiology (Alicante Cohort)

Variables Etiology
Alcoholic Non-alcoholic P
All Patients, N (%) 366 (100) 41 (11.2) 325 (88.8)
Median Age, (IQR) 62 (45-75) 42 (38-48) 65 (49-75) <0.001
Males, N (%) 178 (49) 35 (85) 143 (44) <0.001
BMI≥30, N (%) 94 (26) 5 (12) 89 (27) 0.03
Alcohol Consumption, N (%)
Ever 109 (34) 41 (100) 68 (24) <0.001
Active Smoking, N (%) 90 (28) 31 (79) 59 (21) <0.001
Clinical Outcomes
Any Organ Failure, N(%) 36 (10) 10 (24) 26 (8) 0.001
Persistent Organ Failure, N (%) 17 (5) 4 (10) 13 (4) 0.09
Pancreas Necrosis, N (%)1 46 (17) 15 (38) 31 (14) <0.001
ICU Need 18 (5) 8 (20) 10 (3) <0.001
Median hospitalization (IQR) 11 (7-19) 17 (8-28) 11 (8-18) 0.04
Mortality, N (%) 11 (3) 3 (8) 8 (2) 0.08
1

Contrast enhanced CT was obtained in 265 patients so determination of pancreatic necrosis could only be made in these patients